The Impact of Exercise Intensity on Glycemic Control Following Removal of Exercise
Poster #: 171
Session/Time: B
Author:
Taskina Akhter, MS
Mentor:
Leryn J. Reynolds, PhD
Research Type: Clinical Research
Abstract
INTRODUCTION:
The detrimental effects of exercise cessation on free-living glycemic control (GC) in habitually active individuals are well established. However, the role of exercise intensity to mitigate GC impairment after exercise cessation is not well understood. PURPOSE. To examine if exercise intensity differentially influences the negative impact of exercise cessation on GC.
METHODS:
This randomized, cross over study design recruited nine healthy, recreationally active participants (Age: 26±2 years, BMI: 25.2±1.1 kg/m2, VO2Peak: 39.8±2.0 ml/kg/min) to complete two isocaloric (300 kcals/session) bouts of exercise at moderate (Mod EX: 55-60 %HRR) or vigorous (High EX: 75-80 %HRR) intensity followed by 3 days of exercise removal (No EX). GC was measured via a continuous glucose monitoring system throughout both phases. Other than a standardized breakfast, participants were not provided food. Participants were instructed to consume the same type and volume of food each day across and between phases.
RESULTS:
Physical activity was lower during the No EX phases compared to the exercise phases (Mod EX: 10,714±962 steps/day, 65±7 moderate physical activity (MPA) minutes, 11±4 vigorous physical activity (VPA) minutes; Mod No EX: 5480±817 steps/day, 29±5 MPA minutes, 4±4 VPA minutes), (High EX: 10,246±1021 steps/day, 54±6 MPA minutes, 15±4 VPA minutes, High No EX: 5867±973 steps/day, 34±7 MPA minutes, 4±4 VPA minutes), p<0.05. 24-hour average blood glucose (Mod EX: 90±4, Mod No EX: 94±3, High EX: 87±4, High No EX: 98±3; mg/dL) and average peak post prandial blood glucose (Mod EX: 131±12, Mod No EX: 134±7, High EX: 127±10, High No EX: 141±7; mg/dL) demonstrated no significant main effect of exercise intensity (p>0.05), but did demonstrate a significant main effect of phase (p<0.05).
CONCLUSION:
Removal of exercise for 3 days impairs free-living glycemic control, and exercise intensity does not appear to differentially modulate this response.
The detrimental effects of exercise cessation on free-living glycemic control (GC) in habitually active individuals are well established. However, the role of exercise intensity to mitigate GC impairment after exercise cessation is not well understood. PURPOSE. To examine if exercise intensity differentially influences the negative impact of exercise cessation on GC.
METHODS:
This randomized, cross over study design recruited nine healthy, recreationally active participants (Age: 26±2 years, BMI: 25.2±1.1 kg/m2, VO2Peak: 39.8±2.0 ml/kg/min) to complete two isocaloric (300 kcals/session) bouts of exercise at moderate (Mod EX: 55-60 %HRR) or vigorous (High EX: 75-80 %HRR) intensity followed by 3 days of exercise removal (No EX). GC was measured via a continuous glucose monitoring system throughout both phases. Other than a standardized breakfast, participants were not provided food. Participants were instructed to consume the same type and volume of food each day across and between phases.
RESULTS:
Physical activity was lower during the No EX phases compared to the exercise phases (Mod EX: 10,714±962 steps/day, 65±7 moderate physical activity (MPA) minutes, 11±4 vigorous physical activity (VPA) minutes; Mod No EX: 5480±817 steps/day, 29±5 MPA minutes, 4±4 VPA minutes), (High EX: 10,246±1021 steps/day, 54±6 MPA minutes, 15±4 VPA minutes, High No EX: 5867±973 steps/day, 34±7 MPA minutes, 4±4 VPA minutes), p<0.05. 24-hour average blood glucose (Mod EX: 90±4, Mod No EX: 94±3, High EX: 87±4, High No EX: 98±3; mg/dL) and average peak post prandial blood glucose (Mod EX: 131±12, Mod No EX: 134±7, High EX: 127±10, High No EX: 141±7; mg/dL) demonstrated no significant main effect of exercise intensity (p>0.05), but did demonstrate a significant main effect of phase (p<0.05).
CONCLUSION:
Removal of exercise for 3 days impairs free-living glycemic control, and exercise intensity does not appear to differentially modulate this response.